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What Is A Sleep Apnea?

Sleep apnea is a disorder in which people experience shallow breathing or pauses in breathing during their sleep. African-Americans with moderate or severe sleep apnea are twice as likely to have hard-to-control high blood pressure when their sleep apnea goes untreated, according to a new study funded mainly by the National Heart, Lung, and Blood Institute (NHLBI), part of the National Institutes of Health.
The nonsurgical treatments for obstructive ENT doctor include behavior therapy , including weight loss , medications, dental appliances, continuous positive airway pressure, bi-level positive airway pressure, and auto-titrating continuous positive airway pressure.

This study identifies a risk factor for hard-to-control hypertension that until now has gone underrecognized in African-Americans,” said study leader Dayna Johnson, Ph.D., an assistant professor in the Department of Epidemiology at Rollins School of Public Health at Emory University in Atlanta.
The Inspire Upper Airway System (UAS) is intended for consumers with moderate to severe OSA who have specific characteristics (a Body Mass Index under 32 and the absence of complete collapse in the back of the throat) and were not helped by a CPAP device, or could not tolerate the CPAP treatment.

The Food and Drug Administration ensures the safety and effectiveness of medical devices, including the device most often used by those affected by OSA - the Continuous Positive Airway Pressure machine, commonly known as CPAP - and a new device, the Inspire Upper Airway Stimulation (UAS) System.
These are episodes characterized by mild upper airway narrowing during sleep, with increased respiratory effort required to maintain a slightly reduced level of airflow not large enough to be scored as hypopnea.4 When the increased inspiratory effort required to maintain ventilation is associated with a microarousal, a RERA is scored (Fig.

These craniofacial risk factors have been reported to have their strongest association with sleep apnea in nonobese patients ( 14 ). Several studies have also demonstrated family aggregation of craniofacial morphology (reduction in posterior airway space, increase in mandibular to hyoid distance, inferior hyoid placement) in patients with sleep apnea ( 15 , 16 ). The data from these studies indicate that elements of craniofacial structure in patients with sleep apnea are inherited.

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